freeamfva: Treatment for Chronic Prostatitis

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Treatment for Chronic Prostatitis

We at the MacLeod laboratory examine and treat patients with Urethritis, Acute and Chronic Prostatitis and Infections of the Seminal Vesicles, Epididymis and Testes. Patients seek treatment for symptomatic genital tract infections or for infection caused infertility.To get more news about Seminal vesiculitis treatment, you can visit our official website.
Historical perspectives
Eight years ago The MacLeod Laboratory started offering direct intra-prostatic injections for the treatment of chronic prostatitis.

A retrospective analysis of the data showing the changes in pre and post therapy symptom scores of individual patients is under way. The initial analysis shows very favorable results. I would like to explain the logic behind the procedure and how I took it upon myself as a gynecologist and pathologist to venture into the field of urology and offer an alternative approach to treat one of men’s most troubling and yet most neglected diseases, prostatitis.

During the last five years approximately four hundred males with prostatitis have been treated at The MacLeod Laboratory. A study of these patients and their response to treatment has increased our understanding of the disease itself and the devastating effect chronic prostatitis has on all aspects of a man's life.

Chronic prostatitis can gradually decrease a man’s physical performance and eventually exhaust his mental reserves. His social and sexual life suffer. Bacterial prostatitis can alter or completely impair his partners fertility. Bacteria harbored in the inflamed prostate are transferred into the female's uterus and can cause functional and structural damage to her reproductive tract and interfere with the course of a normal pregnancy. These same bacteria can leave behind life-long stigmata on the newborn.

Even while childbearing is not desired bacteria transferred from an infected prostate can change female pelvic function. The infection alters a woman's hormonal balance, affecting libido and causing other sexual dysfunctions.

As a gynecologist and pathologist I have witnessed the effect of this disease on my female patients who have suffered, sometimes irreversibly from bacteria transmitted by their infected partners. It is clear therefore that my approach to treating prostatitis is drastically different from the isolationist approach of my urologist colleagues. My background as a pathologist first and then a gynecologist– a rare combination in medicine- gives me a unique vantage point. My first fifteen years, after completing medical school, were spent in the laboratory with a microscope, giving me a three dimensional view of diseases and a keen interest in searching for cause and effect correlations in all diseases.

The following thirty years I spent as a practicing clinician in gynecology and obstetrics. My practice was almost exclusively limited to treating infertility and genital tract infections. During these years I became convinced that there are two incomplete branches of medicine, gynecology and urology which should not be studied separately and cannot be understood separately. Males and females are medically interconnected by the fact that sexual intercourse allows not only for reproduction but facilitates the of bacterial flora between the partners.

Due to the profound effect of prostatitis causing bacteria on reproduction in both males and females, they have to be mentioned in the context of reproduction. If you are a young male and you are yet to father a child, prostatitis could affect your fertility and the health of your future children. If you are an older man who was never able to have children, prostatitis could have contributed to your infertility.

Being an infertility specialist and treating men and women for infertility, I have worked closely with urologists. As a pathologist with extensive training in microbiology, it did not take long for me to realize that the overwhelming majority of all infertility cases are infection related. More importantly, the understanding that these infections are shared by both sexes became the paradigm of my medical practice.

While the prostate serves as a sphincter for both the urine and seminal fluid, its more important function is to trap microbes through an elaborate immunological mechanism. The prostate, in response to bacterial invasion produces a host of antibodies. Some of these antibodies are less specific than others and the sperm themselves may get into the crossfire causing anti-sperm antibodies.

This was basically when I began treating the prostate, of asymptomatic infertile males, but later as word traveled — single and married men started to consult me for the sole purpose of receiving therapy for symptomatic prostatitis.

The majority of my patients come to me with the diagnosis of nonbacterial prostatitis. In our laboratory it is exceedingly rare to find negative EPS (expressed prostatic secretion) or semen cultures. Due to lack of final proof we always used the term "association" of a bacterium with certain conditions rather than labeling them as being the cause of that condition. The prescribed antibiotic therapy and the subsequent favorable clinical and reproductive outcome seemed to confirm that we are on the right track. Initially, we treated our patients with usually recommended oral therapy. Our frustration with returning symptoms and positive bacteria findings after repeated oral therapies prompted us to use multi-drug regimens and later intravenously administered antibiotics. Recalling the microscopic image of an enlarged prostate with extensive scarring shielding pockets of bacteria and inflammatory cells, it was easy to understand that oral or even intravenously given antibiotics cannot reach proper concentration in those areas. The introduction of directly injecting antibiotics into the prostate gland delivering 1000 to 2000 fold the concentration of antibiotics than any other previously practiced approach became a logical sequel to our therapy. The injection therapy is rewarded with much improved symptomatic response and great reduction in positive post therapy semen cultures.